###################################### # From Jan Kwasniewski: Homo Optimus # ###################################### TYPE I DIABETES (diabetes mellitus) Type I diabetes is also known as insulin-dependent diabetes mellitus (IDDM) or youth-onset diabetes. There are millions of sufferers from this disease all over the world. These are mainly young people. There are not many old ones. The sufferers don't get a chance to grow old; they die early on in their life. Diabetes shortens life by an average of 30%, markedly lowers the quality of life, costs society dearly, much more than type II diabetes also classified as non insulin-dependent diabetes mellitus (NIDDM). Generally speaking, type I diabetes is not known within the animal kingdom, between both meat- and plant-eating animals, living in their natural habitat. It is however, known to develop in representatives of those species, which due to the lack of knowledge (human) or under the influence of man (dog, domestic pig), are fed a diet suitable only for herbivores or a type of diet, the chemical composition of which makes it inferior to that of herbivores. Type I diabetes does not exist amongst the Eskimo, the Masai, or the Hunza people, and the shepherds of Yakutia, Abkhazia, Georgia or Bulgaria (and many other indigenous people whose diets are typically low in carbohydrate). Type I diabetes is caused by sugar, or to expand on that, a diet, which per 1 g of protein contains more, or far more, carbohydrate than the proportion occurring in the diet of herbivores. In order to metabolise carbohydrate the body needs a lot of protein, vitamins, magnesium and other microelements, which are not contained in sugar, sweetened drinks, honey, potato/maize flour or other pure carbohydrate products. All plant products, which per 1 g of protein contain more than 10 g of carbohydrate, are deficient in those necessary elements. A diet which lacks sufficient amounts of protein forces the body to consume greater amounts of food in order to obtain more protein, vitamins and minerals, but in fact this results in consumption of even more excessive amounts of carbohydrate. Consequently, such a diet lacks the proper amount of protein. It lacks vitamins and magnesium to burn carbohydrate. The body finds a way ;nound the problem by converting a proportion of carbohydrate to fat, thus saving on the expenditure of proteins, vitamins or magnesium. Carbohydrate metabolism, including the processes of conversion to fat and cholesterol, as well as other carbohydrate conversion processes requires insulin. Thus, sugar is the cause of the insulin. When a diet does not contain any carbohydrate, the release of insulin in the human body is minimal. The way in which carbohydrate delivered in food will be metabolised depends on many factors, but mainly on the overall content of the food. And that content influences the proportion of how many grams of sugar are needed to release one unit of insulin. Six grams, 10 g or even 15 g of sugar may be needed to release 1 unit of insulin. The body resists sugar. It defends itself against it. In type I diabetes, this defence consists mainly of destruction of beta cells of the Langerhans islets located in the pancreas, the cells which produce insulin. Destroyed beta cells cannot produce insulin. Several or more months before diabetes develops, the blood of the future diabetic contains antibodies against the beta cells. When the majority of those cells are destroyed the body stops the destruction of the remaining cells. That is why at the time of the first symptoms of the disease antibodies only exist in the blood of approx. 70% patients. The human body will almost never destroy all beta cells. Those that are needed are left intact, not for sugar metabolism but for other important processes. Insulin is needed in the body for a variety of metabolic processes, sugar metabolism being one, although not the most important one. The remaining 5-15% of pancreatic beta cells are still secreting insulin, but that amount is much too small for the large amounts of ingested carbohydrate. However, that amount of insulin would be sufficient if the carbohydrate intake were reduced to match the insulin production by the remaining beta cells. In type I diabetes the blood level of sugar rises because tissues do not require or want any sugar. They defend themselves against the glucose as would the so-called "devil against holy water". Glucose is excreted in the urine; its blood concentration increases dramatically. The suffering individual loses weight because the spare fat is used as fuel in metabolic processes not requiring insulin. The system is overloaded with ketone bodies in amounts well in excess of the body's capacity to burn them. Acidosis 160 161develops. A number of other serious metabolic problems develop. The patient has to die unless insulin is provided. Diabetic patients being "cured" by insulin injections most often die of renal complications caused by pathological changes in the small arterioles of the kidneys, changes that resemble those caused by atherosclerosis. All diets recommended for diabetics during their "convalescence" contain far too much sugar for the amount of insulin still being secreted by the remaining beta cells. Thus, insulin has to be injected. The amount of injected insulin depends on many factors. Sometimes one injection a day is sufficient, at other times a few injections are needed. The body does not want sugar and external insulin and defends itself to its best ability. Antibodies to insulin are produced thus inactivating it. The long-acting types of insulin are not very effective because they are inactivated. The levels of insulin need to be increased to 100 or even 200 units in 24 hours - but the diabetes persists. Surgical removal of the pancreas in carnivores (man, cat, dog, fox, owl, other birds of prey) leads to serious disturbances in their bodies culminating in rapid death. Still, carnivores consume minimal amounts of carbohydrate. Surgical removal of the pancreas in herbivores: the goat, sheep, rabbit, cow, and also ape (!!!), leads to minor disturbances in their metabolism, and these animals, including apes, can survive for a long period of time without the pancreas. Man and the carnivores cannot survive without the pancreas. Insulin is far more essential for man and other carnivores than for herbivores and apes. Carnivores, especially birds of prey, have bigger brains, are far more intelligent than herbivores or seed-eaters, they also live much longer. In terms of anatomy, biochemistry, and histology man is a typical carnivore. Indeed a super-carnivore, taught from the beginning to "enhance" most nutritious, animal-origin food using fire. Humans could not have had a common ancestry with apes because humans cannot survive without the pancreas. Humans have no resistance to African sleeping sickness (trypanosomiasis), to which apes are resistant. Apes, although through necessity, are generally plant eaters, meaning they are unable to obtain food of animal origin or simply have become used to eating plant food, but they resemble carnivores anatomically, more so than do humans. Especially the baboon. Their teeth are typically carnivore-like, but apes. i i i - not carnivores. Human teeth do not at all resemble those of carnivores or herbivores. They are purely human. Our ancestors had the s;ime teeth as we have presently. With one exception, our ancestors did nol generally suffer from tooth decay and their teeth did not fall out because of paradentosis. There were no dentists, because they were not needed. If for those following the optimal diet dentists may initially be iK'cded, later on, however, the need for them will disappear. And so the need for diabetologists will disappear. Chechens do not want Russians (sugar) or their army (insulin). They defend themselves in the same fashion as does the body of the diabetic patient against sugar and insulin. The only solution is to stop the war. When sugar is withdrawn, insulin is not needed. Diabetes will vanish forever. Causal treatment of type I diabetes ŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻ In order to obtain a cure for type I diabetes, the quantity of ingested carbohydrate ought to be reduced to the amount dictated by the insulin-producing capacity of the sufferer. That typically equates with a daily intake of 40 to 50 grams of carbohydrate for an adult, and approx. 1.5 grams per kg of body weight for children. Furthermore, the daily intake of protein should also be reduced to 0.5 grams per kg of body weight, being the highest quality animal-origin protein, e.g., egg yolks, poultry liver, or kidneys. Energy should be mainly sourced from fats of the highest biological value such as bone marrow, egg yolks, cream or butter. The proportions between the nutrients should be maintained at 4 to 5 grams of fat per gram of protein; for the obese that should be reduced to around 2.5 grams of fat. For those who still find it hard to believe that type I diabetes can be successfully cured by the optimal diet I have cited a letter from one of those who experienced "a miracle". I am 17 y.o.; I developed diabetes 2 years ago. My daily dose of insulin was between 15 and 20 units. From the first day on the optimal diet I reduced the insulin dose by half; after a week I stopped it all together. After 6 months on the optimal diet I have no diabetes; other minor health problems are also gone. Maria Nowak (address withheld) Practical guidance ŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻ 1. Implement the optimal diet in its full scope from day 1. 2. Reduce the insulin dose by 50% from day 1. 3. Long-acting insulin should be injected only once a day. If largo doses of insulin are normally required due to inactivation, a few doses of short-acting insulin should be administered. 4. When the requirement for insulin is low ( 1 0 to 15 units/day in adults), injections may be stopped. 5. Blood glucose levels need to be controlled daily. 6. When the fasting blood glucose level falls below 140 mg%, the daily dose of insulin may be reduced by a further 50%. 7. Within a few days for children, or a few weeks for adults, having achieved the fasting blood glucose levels of 140 mg% at a dose of insulin not exceeding 10 to 15 units per day, the insulin should be withdrawn for good. However, that is conditional on stricl adherence to the principles of the optimal diet, as described in this book. Dangers of the transitional period ŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻŻ In theory, a 10- to 15-fold reduction in consumption of carbohydrate should "cure" the diabetes in one day. However, that is not the case. Each cell in the body of a diabetic contains large quantities of many enzymes (proteins) used in the metabolism of sugar. All together the body contains a few kilograms of these enzymes. Following the introduction of the optimal nutrition, over 50% of these now-redundant enzymes are quickly removed (converted) into glucose. One kg of these proteins may produce up to 560 grams of glucose - or a 10- to 12-day allowance of glucose on the optimal diet, not counting the sugar eaten daily. Thus, in the first few days (weeks) following the change-over to the optimal diet most of the sugar comes from within the body rather than from external sources. The quickest cure from diabetes - within a week - can be achieved in children but only in those on low doses of insulin and at the initial stages of the disease. At the latest, the cure will be achieved within 8 to 12 weeks. Between the 6th and 10th day after the introduction of the diet, a transient state of ketoacidosis of variable intensity may develop. This slate is characterised by the appearance of ketone bodies in the urine vviih the intensity being measured using a special cross "+" indicator on ihe scale of I to 4. As mentioned, this is a transient natural state which ulso occurs during fasting. This effect does not indicate that the fasting or the optimal diet ought to be stopped. However, during that stage of I lie diet some temporary modifications should be introduced. Consumption of egg yolks, milk, cream, and vegetables neutralises the acidosis, whereas, egg whites, meat in general (including fish and poultry), and nuts will exacerbate acidosis and therefore should be avoided in the first 10 to 14 days of the diet. A minor temporary increase (10 to 20 grams) in the daily consumption of carbohydrate will also decrease the acidosis. During the first few weeks of the diet carbohydrates should be mainly sourced from vegetables or alternatively from low amounts of fruits which have a low carbohydrate content (see tables at the end of this book). The vegetables can be eaten raw, cooked (from soups), or stewed in butter. After the initial period of two weeks other products containing low amounts of carbohydrate can be included in the menu. The greatest danger of the transitional period may come from hypoglycaemia when the blood glucose level falls below 50 mg% as a result of an insulin overdose. Therefore, during that period one should ensure that the level of glucose does not fall below 90 to 100 mg%. Thus, it is preferable to slightly under-dose the insulin than to over-dose it. Just in case, one should carry a sweet or a sachet of sugar at all times. Unstable type I diabetes, with complications and requiring large doses of insulin, or that affecting young children should always be treated in a hospital setting. However, that of course creates problems since the medical profession and especially diabetes specialists firmly believe that type I diabetes is incurable. Having said that, the combination of an appropriate level of intelligence (of parents in the case of a diabetic child) with strict adherence to the diet (and good knowledge of the chemical content of products), and with a recently-acquired or an uncomplicated and stable disease (requiring low doses of insulin) should result in successful treatment at home. However, that form of treatment ought to be an exception rather than a common occurrence. Once the cure has been achieved, the reader is encouraged to forward his/her personal details to the address provided at the end ol this book, together with the description of the disease (the date of the onset, the dose and type of insulin), the dose of insulin after the introduction of the diet and the length of period before withdrawal, as well as the length of the time needed to stabilise the blood glucose level.